This is a preprint, a preliminary version of a manuscript that has not completed peer review at a journal. Research Square does not conduct peer review prior to posting preprints. The posting of a preprint on this server should not be interpreted as an endorsement of its validity or suitability for dissemination as established information or for guiding clinical practice.
Case report
Qian Zhang
Hebei Medical University Fourth Affiliated Hospital and Hebei Provincial Tumor Hospital
Corresponding Author
ORCiD: https://orcid.org/0000-0002-7742-8393
License:
This work is licensed under a CC BY 4.0 License. Read Full License
Background Post-diuretic pulmonary edema(PDPE)is a special type of acute pulmonary edema, results from rapid blood volume loss caused by diuretics. Inappropriate administration of diuretics and inotropic agent, which is mainstay of treatment of pulmonary edema Classically, might have been a critical mistake in PDPE.Maintaining appropriate volume status and beta receptor blockers is the key to reversing the progress of PDPE. However,this condition remains unfamiliar to the medical community, making it an underdiagnosed and underreported condition. Case representation A 71-year-old patient who initially was diagnosed to have acute respiratory distress syndrome (ADRS) in intensive care unit (ICU) but got further dyspnea after rapid volume depletion. Point of care ultrasound (POCUS) examination showed worsened pulmonary edema with inferior vena cava collapsed during inspiratory period and a small but hyperdynamic left ventricular(LV). The patient subsequently improved with fluid, beta blocker, increased sedation, and paralysis. Conclusion We first put forward the concept of post-diuretic pulmonary edema,in order to remind medical staff to pay attention to this type of pulmonary edema and understand its pathophysiological mechanism.
Keywords
Pulmonary edema, diuretic, ultrasound
Figure 1
Figure 2
Figure 3
Figure 4
This is a list of supplementary files associated with this preprint. Click to download.
Loading...
Badges
Prescreen
This manuscript passed our Prescreen assessment
Complete author information
Appropriate declaration statements
Potential risks to human health
Prescreen
History
CURRENT STATUS: Posted
Version 1
Posted 15 Mar, 2019
No community comments so far
Metrics
Comments: 0
PDF Downloads: ...
HTML Views: ...
Subject Areas
Internal Medicine Specialties
Learn more about our company.
This is a preprint. It has not completed peer review.
Case report
Qian Zhang
Hebei Medical University Fourth Affiliated Hospital and Hebei Provincial Tumor Hospital
Corresponding Author
ORCiD: https://orcid.org/0000-0002-7742-8393
Badges
Prescreen
This manuscript passed our Prescreen assessment
Complete author information
Appropriate declaration statements
Potential risks to human health
Prescreen
History
CURRENT STATUS: Posted
Version 1
Posted 15 Mar, 2019
No community comments so far
Metrics
Comments: 0
PDF Downloads: ...
HTML Views: ...
Subject Areas
Internal Medicine Specialties
License:
This work is licensed under a CC BY 4.0 License. Read Full License
Background Post-diuretic pulmonary edema(PDPE)is a special type of acute pulmonary edema, results from rapid blood volume loss caused by diuretics. Inappropriate administration of diuretics and inotropic agent, which is mainstay of treatment of pulmonary edema Classically, might have been a critical mistake in PDPE.Maintaining appropriate volume status and beta receptor blockers is the key to reversing the progress of PDPE. However,this condition remains unfamiliar to the medical community, making it an underdiagnosed and underreported condition. Case representation A 71-year-old patient who initially was diagnosed to have acute respiratory distress syndrome (ADRS) in intensive care unit (ICU) but got further dyspnea after rapid volume depletion. Point of care ultrasound (POCUS) examination showed worsened pulmonary edema with inferior vena cava collapsed during inspiratory period and a small but hyperdynamic left ventricular(LV). The patient subsequently improved with fluid, beta blocker, increased sedation, and paralysis. Conclusion We first put forward the concept of post-diuretic pulmonary edema,in order to remind medical staff to pay attention to this type of pulmonary edema and understand its pathophysiological mechanism.
Figure 1
Figure 2
Figure 3
Figure 4
Loading...