[1] H. Izumi, M. Tsuda, Y. Sato et al. “Bovine milk exosomes contain microRNA and mRNA and are taken up by human macrophages”. Journal Dairy Science, 98(5):2920-33, 2015.
[2] T. Wolf, S.R. Baier, J. Zempleni. “The Intestinal Transport of Bovine Milk Exosomes Is Mediated by Endocytosis in Human Colon Carcinoma Caco-2 Cells and Rat Small Intestinal IEC-6 Cells”. Journal of Nutrition. 145(10):2201-6, 2015.
[3] S.L. Ong, C. Blenkiron, S. Haines et al. “Ruminant Milk-Derived Extracellular Vesicles: A Nutritional and Therapeutic Opportunity?” Nutrients. 22;13(8):2505, 2021.
[4] S. Pal, K. Woodford, S. Kukuljan, S. Ho. “Milk Intolerance, Beta-Casein and Lactose”. Nutrients. 31;7(9):7285-97, 2015
[5] X. Wang, X. Kong, Y. Qin et al. “Milk phospholipids ameliorate mouse colitis associated with colonic goblet cell depletion via the Notch pathway”. Food Function. 1;10(8):4608-4619, 2019.
[6] J.W. Crayton. “Adverse reactions to foods: relevance to psychiatric disorders”. J Allergy Clin Immunol. 78(1 Pt 2):243-50, 1986.
[7] N.A. Smith, D.L. Germundson, P. Gao et al. “Anxiety-like behavior and intestinal microbiota changes as strain-and sex-dependent sequelae of mild food allergy in mouse models of cow's milk allergy”. Brain Behavior Immunity. 95:122-141, 2021.
[8] R. Chunder, A. Weier, H. Mäurer et al. “Antibody cross-reactivity between casein and myelin-associated glycoprotein results in central nervous system demyelination”. Proc Natl Acad Sci USA. 8;119(10):e2117034119, 2022.
[9] S. Kippenberger, N. Zöller, J. Kleemann et al. “STAT6-Dependent Collagen Synthesis in Human Fibroblasts Is Induced by Bovine Milk”. PLoS One. 2;10(7):e0131783, 2015.
[10] S. Manca, B. Upadhyaya, E. Mutai et al. “Milk exosomes are bioavailable and distinct microRNA cargos have unique tissue distribution patterns”. Scientific Reports. 27;8(1):11321, 2018.
[11] T.M. Nordgren, A.J. Heires, J. Zempleni et al. “Bovine milk-derived extracellular vesicles enhance inflammation and promote M1 polarization following agricultural dust exposure in mice”. Journal Nutr Biochem. 64:110-120, 2019.
[12] E.S. Brodkin. “BALB/c mice: low sociability and other phenotypes that may be relevant to autism”. Behav Brain Res. 10;176(1):53-65, 2007.
[13] R. Mondragón, L. Mayagoitia, A. López-Luján, J.L. Díaz. “Social structure features in three inbred strains of mice, C57Bl/6J, Balb/cj, and NIH: a comparative study”. Behav Neural Biol. 47(3):384-91, 1987.
[14] J. Peça, C. Feliciano, J.T. Ting et al. “Shank3 mutant mice display autistic-like behaviours and striatal dysfunction”. Nature. 28;472(7344):437-42, 2011.
[15] J. Borovac, M. Bosch, K. Okamoto. “Regulation of actin dynamics during structural plasticity of dendritic spines: Signaling messengers and actin-binding proteins”. Mol Cell Neuroscience. 91:122-130, 2018.
[16] D.M. Basso, L.C. Fisher, A.J. Anderson et al. “Basso Mouse Scale for locomotion detects differences in recovery after spinal cord injury in five common mouse strains”. J Neurotrauma. 23(5):635-59, 2006.
[17] L.P. Diniz, J.C. Almeida, V. Tortelli et al. “Astrocyte-induced synaptogenesis is mediated by transforming growth factor β signaling through modulation of D-serine levels in cerebral cortex neurons”. J Biol Chem. 30;287(49):41432-45, 2012.
[18] Q. Chen, C.A. Deister, X. Gao et al. “Dysfunction of cortical GABAergic neurons leads to sensory hyper-reactivity in a Shank3 mouse model of ASD”. Nature Neuroscience. 23(4):520-532, 2020.
[19] X. Liao, J. Yang, H. Wang, Y. Li. “Microglia mediated neuroinflammation in autism spectrum disorder”. J Psychiatr Res. 130:167-176, 2020.
[20] S.M. Matta, E.L. Hill-Yardin, P.J Crack. “The influence of neuroinflammation in Autism Spectrum Disorder”. Brain Behav Immun. 79:75-90, 2019.
[21] E.M. Sajdel-Sulkowska, M. Xu, N. Koibuchi. “Increase in cerebellar neurotrophin-3 and oxidative stress markers in autism”. Cerebellum. 8(3):366-72, 2009.
[22] N.M.K. Bruchhage, M.P. Bucci, E.B.E. Becker. “Cerebellar involvement in autism and ADHD”. Handb Clin Neurol. 155:61-72, 2018.
[23] D. Turck. “Cow's milk and goat's milk”. World Rev Nutr Diet. 108:56-62, 2013.
[24] H. Cekici, N. Sanlier. “Current nutritional approaches in managing autism spectrum disorder: A review”. Nutr Neurosci. 22(3):145-155, 2019.
[25] P. Monteiro, G. Feng. “SHANK proteins: roles at the synapse and in autism spectrum disorder”. Nature Reviews Neuroscience. 18(3):147-157, 2017.
[26] E.C. Cope, B.A. Briones, A.T. Brockett et al. “Immature Neurons and Radial Glia, But Not Astrocytes or Microglia, Are Altered in Adult Cntnap2 and Shank3 Mice, Models of Autism”. eNeuro. 17;3(5):ENEURO.0196-16.2016, 2016.
[27] L. Qin, K. Ma, Z.J. Wang et al. “Social deficits in Shank3-deficient mouse models of autism are rescued by histone deacetylase (HDAC) inhibition”. Nature Neuroscience. 21(4):564-575, 2018.
[28] H. Takahashi, T. Mizui, T. Shirao. “Down-regulation of drebrin A expression suppresses synaptic targeting of NMDA receptors in developing hippocampal neurons”. J Neurochem. 97 Suppl 1:110-5, 2006.
[29] M.A. Calcia, D.R. Bonsall, P.S. Bloomfield et al. “Stress and neuroinflammation: a systematic review of the effects of stress on microglia and the implications for mental illness”. Psychopharmacology (Berl). 233(9):1637-50, 2016.
[30] K. Borst, A.A. Dumas, M. Prinz. “Microglia: Immune and non-immune functions”. Immunity. 12;54(10):2194-2208, 2021.
[31] F. Petrelli, L. Pucci, P. Bezzi. “Astrocytes and Microglia and Their Potential Link with Autism Spectrum Disorders”. Front Cell Neuroscience. 12;10:21, 2016.
[32] L.S. Abdelli, A. Samsam, S.A Naser. “Propionic Acid Induces Gliosis and Neuro-inflammation through Modulation of PTEN/AKT Pathway in Autism Spectrum Disorder”. Scientific Reports. 19;9(1):8824, 2019.
[33] J. Ellegood, J.N. Crawley. “Behavioral and Neuroanatomical Phenotypes in Mouse Models of Autism. Neurotherapeutics”. 12(3):521-33, 2015.
[34] D.T. Stephenson, S.M. O'Neill, S. Narayan et al. “Histopathologic characterization of the BTBR mouse model of autistic-like behavior reveals selective changes in neurodevelopmental proteins and adult hippocampal neurogenesis”. Mol Autism. 16;2(1):7, 2011.
[35] Q. Zhang, H. Wu, M. Zou et al., “Folic acid improves abnormal behavior via mitigation of oxidative stress, inflammation, and ferroptosis in the BTBR T+ tf/J mouse model of autism”. J Nutr Biochem. 71:98-109, 2019.
[36] A. Nadeem, S.F. Ahmad, N.O. Al-Harbi et al. “Increased oxidative stress in the cerebellum and peripheral immune cells leads to exaggerated autism-like repetitive behavior due to deficiency of antioxidant response in BTBR T + tf/J mice”. Prog Neuropsychopharmacol Biol Psychiatry. 8;89:245-253, 2019.
[37] I.A. Lopez, D. Acuna, L. Beltran-Parrazal et al. “Evidence for oxidative stress in the developing cerebellum of the rat after chronic mild carbon monoxide exposure (0.0025% in air)”. BMC Neuroscience. 27;10:53, 2009.
[38] J. Rodrigo, D. Alonso, A.P. Fernández et al. “Neuronal and inducible nitric oxide synthase expression and protein nitration in rat cerebellum after oxygen and glucose deprivation”. Brain Res. 3;909(1-2):20-45, 2001.
[39] Lemos et al. Pre-print: https://www.researchsquare.com/article/rs-141155/v1.