[1] G. Zhiqiang, D. Zhaohui, W. Qinhuan et al., “Cost of chronic hepatitisB infection in China,”Journal of Clinical Gastroenterology, vol. 38, supplement 3, no. 10, pp. S175–S178, 2004.
[2] B. Combes, J. Shorey, A. Barrera et al., “Glomerulonephritis with deposition of Australia antigen-antibody complexes in glomerular basement membrane,” The Lancet, vol. 2, no. 7718, pp.234–237, 1971.
[3] W. G. Couser and D. J. Salant, “In situ immune complex formation and glomerular injury,” Kidney International, vol. 17,no. 1, pp. 1–13, 1980.e
[4] Almine, J.F., O’Hare, C.A., Dunphy, G., Haga, I.R., Naik, R.J., Atrih, A., Connolly, D.J., Taylor, J., Kelsall, I.R., Bowie, A.G., et al. (2017). IFI16 and cGAS cooperate in the activation of STING during DNA sensing in human keratinocytes. Nat. Commun. 8, 14392.
[5] Goubau, D., Deddouche, S., and Reis e Sousa, C. (2013). Cytosolic sensing of viruses. Immunity 38, 855–869
[6] Ansari MA, Dutta S, Veettil MV, Dutta D, Iqbal J, Kumar B, Roy A, Chikoti L, Singh VV, Chandran B. 2015. Herpesvirus genome recognition induced acetylation of nuclear IFI16 is essential for its cytoplasmic translocation, inflammasome and IFN-b responses. PLOS Pathogens 11:e1005019.
[7] Kerur N, Veettil MV, Sharma-Walia N, Bottero V, Sadagopan S, Otageri P, Chandran B. 2011. IFI16 acts as a nuclear pathogen sensor to induce the inflammasome in response to kaposi Sarcoma-associated herpesvirus infection. Cell Host & Microbe 9:363–375.
[8] Unterholzner L, Keating SE, Baran M, Horan KA, Jensen SB, Sharma S, Sirois CM, Jin T, Latz E, Xiao TS, Fitzgerald KA, Paludan SR, Bowie AG. 2010. IFI16 is an innate immune sensor for intracellular DNA. Nature Immunology 11:997–1004
[9] Dunphy, G., Flannery, S.M., Almine, J.F., Connolly, D.J., Paulus, C., Jonsson, K.L., Jakobsen, M.R., Nevels, M.M., Bowie, A.G., and Unterholzner, L. (2018). Non-canonical Activation of the DNA Sensing Adaptor STING by ATM and IFI16 Mediates NF-kappaB Signaling after Nuclear DNA Damage. Mol. Cell 71, 745–760.e5.
[10] Biggioggero, M., Gabbriellini, L., and Meroni, P.L. (2010). Type I interferon therapy and its role in autoimmunity. Autoimmunity 43, 248–254.
[11] Iwamoto, S., Kido, M., Aoki, N., Nishiura, H., Maruoka, R., Ikeda, A., Okazaki, T., Chiba, T., and Watanabe, N. (2012). IFN-g is reciprocally involved in the concurrent development of organ-specific autoimmunity in the liver and stomach. Autoimmunity 45, 186–198.
[12] He XY, Fang LJ, Zhang YE, Sheng FY, Zhang XR, Guo MY: In situ hybridization of hepatitis B DNA in hepatitis B-associated glomerulonephritis. Pediatr Nephrol 1998, 12:117–120.
[13] K/DOQI clinical practice guidelines for chronic kidney disease: evaluation, classification, and stratification, American journal of kidney diseases, K/DOQI clinical practice guidelines for chronic kidney disease: Evaluation, classification, and stratification, American journal of kidney diseases. J National Kidney 2002, 39:S1–266
[14] Lee HS, Choi Y, Yu SH, Koh HI, Kim MJ, Ko KW: A renal biopsy study of hepatitis B virus-associated nephropathy in Korea. Kidney Int 1988, 34:537–543.
[15] Lin C-Y, Lin C-C, Chang G-J, King C-C: Defect of Cell-Mediated Immune Response against Hepatitis B Virus; An Indication for Pathogenesis of Hepatitis-B-Virus-Associated Membranous Nephropathy. Nephron 1997, 76:176-185.
[16] Lin C-Y, Lin C-C, Chang G-J, King C-C: Defect of Cell-Mediated Immune Response against Hepatitis B Virus; An Indication for Pathogenesis of Hepatitis-B-Virus-Associated Membranous Nephropathy. Nephron 1997, 76:176-185.
[17] Kusakabe A, Tanaka Y, Kurbanov F, Goto K, Tajiri H, Murakami J, Okuse C, Yotsuyanagi H, Joh T, Mizokami M: Virological features of hepatitis B virus‐associated nephropathy in Japan. Journal of medical virology 2007, 79:1305-1311.
[18] Ren J, Wang L, Chen Z, Ma Z, Zhu H, Yang D, Li X, Wang B, Fei J, Wang Z: Gene expression profile of transgenic mouse kidney reveals pathogenesis of hepatitis B virus associated nephropathy. Journal of medical virology 2006, 78:551-560
[19] Wang N-S, Wu Z-L, Zhang Y-E, Liao L-T: Existence and significance of hepatitis B virus DNA in kidneys of IgA nephropathy. World J Gastroenterol 2005, 11:712-716.
[20] Deng C-L, Song X-W, Liang H-J, Chen F, Sheng Y-J, Wang M-Y: Chronic hepatitis B serum promotes apoptotic damage in human renal tubular cells. World Journal of Gastroenterology 2006, 12:1752
[21] M. Bortolami, A. Kotsafti, R. Cardin, and F. Farinati, “Fas/FasL system, IL-1ß expression and apoptosis in chronic HBV and HCV liver diseases. Journal of Viral Hepatitis, vol. 15, no. 7, pp. 515–522, 2008.