Barlow, B.K., Thiruchelvam, M.J., Bennice, L., Cory-Slechta, D.A., Ballatori, N., et al, 2003. Increased synaptosomal dopamine content and brain concentration of paraquat produced by selective dithiocarbamates. Journal of Neurochemistry. 85, 1075-1086. https://doi.org/10.1046/j.1471-4159.2003.01773.x.
Bonneh-Barkay, D., Langston, W.J., Di Monte, D.A., 2005. Toxicity of redox cycling pesticides in primary mesencephalic cultures. Antioxid Redox Signal. 7, 649-653. https://doi.org/10.1089/ars.2005.7.649.
Brooks, A.I., Chadwick, C.A., Gelbard, H.A., Cory-Slechta, D.A., Federoff, H.J., 1999. Paraquat elicited neurobehavioral syndrome caused by dopaminergic neuron loss. Brain research. 823, 1-10. https://doi.org/10.1016/s0006-8993(98)01192-5.
Cassar, M., Issa, A.R., Riemensperger, T., Petitgas, C., Rival, T., et al, 2015. A dopamine receptor contributes to paraquat-induced neurotoxicity in Drosophila. Human molecular genetics. 24, 197-212. https://doi.org/10.1093/hmg/ddu430.
Chen, L., Na, R., Boldt, E., Ran, Q., 2015. NLRP3 inflammasome activation by mitochondrial reactive oxygen species plays a key role in long-term cognitive impairment induced by paraquat exposure. Neurobiology of aging. 36, 2533-2543. https://doi.org/10.1016/j.neurobiolaging.2015.05.018.
Cherry, J.D., Olschowka, J.A., O'Banion, M.K., 2014. Neuroinflammation and M2 phenotype: the good, the bad, and the inflamed. Journal of neuroinflammation. 11, 98. https://doi.org/10.1186/1742-2094-11-98.
Chhor, V., Le Charpentier, T., Lebon, S., Oré, M.V., Celador, I.L., et al, 2013. Characterization of phenotype markers and neuronotoxic potential of polarised primary microglia in vitro. Brain, behavior, and immunity. 32, 70-85. https://doi.org/10.1016/j.bbi.2013.02.005.
Crain, J.M., Nikodemova, M., Watters, J.J., 2013. Microglia express distinct M1 and M2 phenotypic markers in the postnatal and adult central nervous system in male and female mice. Journal of neuroscience research. 91, 1143–1151. https://doi.org/10.1002/jnr.23242.
Jha, M.K., Lee, W.H., Suk, K., 2016. Functional polarization of neuroglia: Implications in neuroinflammation and neurological disorders. Biochemical pharmacology. 103, 1-16. https://doi.org/10.1016/j.bcp.2015.11.003.
Kadowaki, T., Nakadate, K., Sakakibara, S., Hirata, K., Ueda, S., 2007. Expression of Iba1 protein in microglial cells of zitter mutant rat. Neuroscience letters. 411, 26-31. https://doi.org/10.1016/j.neulet.2006.07.079.
Kim, S., Hwang, J., Lee, W.H., Hwang, D.Y., Suk, K., 2008. Role of protein kinase Cdelta in paraquat-induced glial cell death. Journal of neuroscience research. 86, 2062-2070. https://doi.org/10.1002/jnr.21643.
Lawson, L.J., Perry, V.H., Dri, P., Gordon, S., 1990. Heterogeneity in the distribution and morphology of microglia in the normal adult mouse brain. Neuroscience. 39, 151–170. https://doi.org/10.1016/0306-4522(90)90229-w.
Mantovani, A., Sica, A., Sozzani, S., Allavena, P., Vecchi, A., Locati, M., 2004. The chemokine system in diverse forms of macrophage activation and polarization. Trends in immunology. 25, 677-686. https://doi.org/10.1016/j.it.2004.09.015.
McCormack, A.L., Thiruchelvam, M., Manning-Bog, A.B., Thiffault, C., Langston, J.W., et al, 2002. Environmental Risk Factors and Parkinson's Disease: Selective Degeneration of Nigral Dopaminergic Neurons Caused by the Herbicide Paraquat. Neurobiology of Disease. 10,119-127. https://doi.org/10.1006/nbdi.2002.0507.
Mouton, P.R., Long, J.M., Lei, D..L, Howard ,V., Jucker, M., et al, 2002. Age and gender effects on microglia and astrocyte numbers in brains of mice. Brain research. 956, 30–35. https://doi.org/10.1016/s0006-8993(02)03475-3.
Nakagawa, Y., Chiba, K., 2014. Role of microglial m1/M2 phenotype in relapse and remission of psychiatric disorders and diseases. Pharmaceuticals (Basel, Switzerland). 7, 1028-1048. https://doi.org/10.3390/ph7121028.
Schindelin, J., Arganda-Carreras, I., Frise, E., Kaynig, V., Longair, M., et al, 2012. Fiji: an open-source platform for biological-image analysis. Nature methods. 9, 676-682. https://doi.org/10.1038/nmeth.2019.
Shimizu, K., Ohtaki, K., Matsubara, K., Aoyama, K., Uezono, T., et al, 2001. Carrier-mediated processes in blood--brain barrier penetration and neural uptake of paraquat. Brain research. 906, 135-142. https://doi.org/10.1016/s0006-8993(01)02577-x.
Sun, L., Yan, P.B., Zhang, Y.,Wei, L. Q., Li, G.Q., 2018. Effect of activated charcoal hemoperfusion on renal function in patients with paraquat poisoning. Experimental and therapeutic medicine. 15, 2688-2692. https://doi.org/10.3892/etm.2018.5712.
Wu, B., Song, B., Tian, S., Huo, S., Cui, C., et al, 2012. Central nervous system damage due to acute paraquat poisoning: a neuroimaging study with 3.0 T MRI. Neurotoxicology. 33, 1330-1337. https://doi.org/10.1016/j.neuro.2012.08.007.
Wu, B., Song, B., Yang, H., Huang, B., Chi, B., et al, 2013. Central nervous system damage due to acute paraquat poisoning: an experimental study with rat model. Neurotoxicology. 35, 62-70. https://doi.org/10.1016/j.neuro.2012.12.001.
Wu, B.L., Song, B., 2013a. Reply to Dr. Jeffrey Brent. Neurotoxicology. 37, 220. https://doi.org/10.1016/j.neuro.2013.05.013.
Wu, B.L., Song, B., 2013b. Reply to drs. John andrew tomenson and clive campbell. Neurotoxicology. 36, 105. https://doi.org/10.1016/j.neuro.2013.02.011.
Wu, X.F., Block, M.L., Zhang, W., Qin, L., Wilson, B., et al, 2005. The role of microglia in paraquat-induced dopaminergic neurotoxicity. Antioxid Redox Signal. 7, 654-661. https://doi.org/10.1089/ars.2005.7.654.
Young, K., Morrison, H., 2018. Quantifying Microglia Morphology from Photomicrographs of Immunohistochemistry Prepared Tissue Using ImageJ. Journal of visualized experiments : JoVE. 136,57648. https://doi.org/10.3791/57648.
Zhang, X.F., Thompson, M., Xu, Y.H., 2016. Multifactorial theory applied to the neurotoxicity of paraquat and paraquat-induced mechanisms of developing Parkinson's disease. Laboratory investigation; a journal of technical methods and pathology. 96, 496-507. https://doi.org/10.1038/labinvest.2015.161.