1.Bray, F., et al., Global estimates of cancer prevalence for 27 sites in the adult population in 2008. Int J Cancer, 2013. 132(5): p. 1133–45.
2.Bertuccio, P., et al., Recent patterns in gastric cancer: a global overview. Int J Cancer, 2009. 125(3): p. 666–73.
3.Sigon, R., V. Canzonieri, and C. Rossi, Early gastric cancer: a single-institution experience on 60 cases. Suppl Tumori, 2003. 2(5): p. S23–6.
4.Chen, W., et al., Cancer statistics in China, 2015. CA Cancer J Clin, 2016. 66(2): p. 115–32.
5.Ferlay, J., et al., Cancer incidence and mortality worldwide: sources, methods and major patterns in GLOBOCAN 2012. Int J Cancer, 2015. 136(5): p. E359–86.
6.<Continuous Update Project Report Diet, Nutrition, Physical Activity and Stomach Cancer. 2016.pdf>.
7.Cheng, X. J., J. C. Lin, and S. P. Tu, Etiology and Prevention of Gastric Cancer. Gastrointest Tumors, 2016. 3(1): p. 25–36.
8.Correa, P. and M. B. Piazuelo, Helicobacter pylori Infection and Gastric Adenocarcinoma. US Gastroenterol Hepatol Rev, 2011. 7(1): p. 59–64.
9.Wroblewski, L. E., R. M. Peek, and K. T. Wilson, Helicobacter pylori and Gastric Cancer: Factors That Modulate Disease Risk. Clin Microbiol Rev, 2010. 23(4): p. 713–39.
10.Polk, D. B. and R. M. Peek, Helicobacter pylori: gastric cancer and beyond. Nat Rev Cancer, 2010. 10(6): p. 403–14.
11.Schistosomes, liver flukes and Helicobacter pylori. IARC Working Group on the Evaluation of Carcinogenic Risks to Humans. Lyon, 7–14 June 1994. IARC Monogr Eval Carcinog Risks Hum, 1994. 61: p. 1–241.
12.Shiota, S., et al., Systematic review and meta-analysis: the relationship between the Helicobacter pylori dupA gene and clinical outcomes. Gut Pathog, 2010. 2(1): p. 13.
13.Costa, A. C., C. Figueiredo, and E. Touati, Pathogenesis of Helicobacter pylori infection. Helicobacter, 2009. 14 Suppl 1: p. 15–20.
14.Sahara, S., et al., Role of Helicobacter pylori cagA EPIYA motif and vacA genotypes for the development of gastrointestinal diseases in Southeast Asian countries: a meta-analysis. BMC Infect Dis, 2012. 12: p. 223.
15.Azevedo, N. F., et al., A new model for the transmission of Helicobacter pylori: role of environmental reservoirs as gene pools to increase strain diversity. Crit Rev Microbiol, 2007. 33(3): p. 157–69.
16.Latifi-Navid, S., et al., Helicobacter pylori vacA d1/-i1 genotypes and geographic differentiation between high and low incidence areas of gastric cancer in Iran. Arch Iran Med, 2013. 16(6): p. 330–7.
17.Con, S. A., et al., Role of bacterial and genetic factors in gastric cancer in Costa Rica. World J Gastroenterol, 2009. 15(2): p. 211–8.
18.Ilver, D., et al., Helicobacter pylori adhesin binding fucosylated histo-blood group antigens revealed by retagging. Science, 1998. 279(5349): p. 373–7.
19.Boren, T., et al., Attachment of Helicobacter pylori to human gastric epithelium mediated by blood group antigens. Science, 1993. 262(5141): p. 1892–5.
20.Pride, D. T., R. J. Meinersmann, and M. J. Blaser, Allelic Variation within Helicobacter pylori babA and babB. Infect Immun, 2001. 69(2): p. 1160–71.
21.Chen, M. Y., et al., Association of Helicobacter pylori babA2 with peptic ulcer disease and gastric cancer. World J Gastroenterol, 2013. 19(26): p. 4242–51.
22.Roman-Roman, A., et al., Helicobacter pylori vacA s1m1 genotype but not cagA or babA2 increase the risk of ulcer and gastric cancer in patients from Southern Mexico. Gut Pathog, 2017. 9: p. 18.
23.Mottaghi, B., et al., Helicobacter pylori vacA i region polymorphism but not babA2 status associated to gastric cancer risk in northwestern Iran. Clin Exp Med, 2016. 16(1): p. 57–63.
24.Abdi, E., et al., Helicobacter pylori babA2 Positivity Predicts Risk of Gastric Cancer in Ardabil, a Very High-Risk Area in Iran. Asian Pac J Cancer Prev, 2016. 17(2): p. 733–8.
25.Talebi Bezmin Abadi, A., et al., High correlation of babA 2-positive strains of Helicobacter pylori with the presence of gastric cancer. Intern Emerg Med, 2013. 8(6): p. 497–501.
26.Saxena, A., et al., Virulence attributes of Helicobacter pylori isolates & their association with gastroduodenal disease. Indian J Med Res, 2011. 133: p. 514–20.
27.Mattar, R., et al., Association of LEC and tnpA Helicobacter pylori genes with gastric cancer in a Brazilian population. Infect Agent Cancer, 2010. 5: p. 1.
28.Ghasemian Safaei, H., et al., Relation of bab A2 genotype of Helicobacter pylori infection with chronic active gastritis, duodenal ulcer and non-cardia active gastritis in Alzahra hospital Isfahan, Iran. Jundishapur J Microbiol, 2010. 3(3): p. 93–98.
29.Bartchewsky, W., Jr., et al., Effect of Helicobacter pylori infection on IL–8, IL–1beta and COX–2 expression in patients with chronic gastritis and gastric cancer. Scand J Gastroenterol, 2009. 44(2): p. 153–61.
30.Zhang, Z., et al., The Helicobacter pylori duodenal ulcer promoting gene, dupA in China. BMC Gastroenterol, 2008. 8: p. 49.
31.Erzin, Y., et al., Role of host interleukin 1beta gene (IL–1B) and interleukin 1 receptor antagonist gene (IL–1RN) polymorphisms in clinical outcomes in Helicobacter pylori-positive Turkish patients with dyspepsia. J Gastroenterol, 2008. 43(9): p. 705–10.
32.Chomvarin, C., et al., Prevalence of Helicobacter pylori vacA, cagA, cagE, iceA and babA2 genotypes in Thai dyspeptic patients. Int J Infect Dis, 2008. 12(1): p. 30–6.
33.Lee, H. S., et al., Expression of Lewis antigens and their precursors in gastric mucosa: relationship with Helicobacter pylori infection and gastric carcinogenesis. J Pathol, 2006. 209(1): p. 88–94.
34.Han, Y. H., et al., Clinical relevance of iceA and babA2 genotypes of Helicobacter pylori in a Shanghai population. Chin J Dig Dis, 2004. 5(4): p. 181–5.
35.Oliveira, A. G., et al., babA2- and cagA-positive Helicobacter pylori strains are associated with duodenal ulcer and gastric carcinoma in Brazil. J Clin Microbiol, 2003. 41(8): p. 3964–6.
36.Yamaoka, Y., et al., Importance of Helicobacter pylori oipA in clinical presentation, gastric inflammation, and mucosal interleukin 8 production. Gastroenterology, 2002. 123(2): p. 414–24.
37.Mizushima, T., et al., Clinical relevance of the babA2 genotype of Helicobacter pylori in Japanese clinical isolates. J Clin Microbiol, 2001. 39(7): p. 2463–5.
38.Gerhard, M., et al., Clinical relevance of the Helicobacter pylori gene for blood-group antigen-binding adhesin. Proc Natl Acad Sci U S A, 1999. 96(22): p. 12778–83.
39.Wells, G., et al., The Newcastle-Ottawa Scale (NOS) for assessing the quality of case-control studies in meta-analyses. Vol. 25. 2011. 603–605.
40.Higgins, J. P. and S. G. Thompson, Quantifying heterogeneity in a meta-analysis. Stat Med, 2002. 21(11): p. 1539–58.
41.Cochran, W. G., The Combination of Estimates from Different Experiments. Biometrics, 1954. 10(1): p. 101–129.
42.DerSimonian, R. and N. Laird, Meta-analysis in clinical trials. Control Clin Trials, 1986. 7(3): p. 177–88.
43.Egger, M., et al., Bias in meta-analysis detected by a simple, graphical test. BMJ, 1997. 315(7109): p. 629–34.
44.pormohammad, A., et al., Risk of gastric cancer in association with Helicobacter pylori different virulence factors: A systematic review and meta-analysis. Microbial Pathogenesis, 2018. 118: p. 214–219.
45.Zagari, R. M. and F. Bazzoli, Gastric cancer: who is at risk? Dig Dis, 2004. 22(4): p. 302–5.
46.Maeda, S., et al., Relationship between nuclear factor-kappaB activation and virulence factors of Helicobacter pylori in Japanese clinical isolates. J Gastroenterol Hepatol, 2002. 17(5): p. 556–62.
47.Heidari, K., et al., The Prevalence of Helicobacter pylori Virulence Related Genes (hpa and babA2) in Iranian Patients with Gastrointestinal Disorders. Jundishapur J Microbiol, 2017. 10(12): p. e60947.
48.Yu, J., et al., Relationship between Helicobacter pylori babA2 status with gastric epithelial cell turnover and premalignant gastric lesions. Gut, 2002. 51(4): p. 480–4.